the several thin layers of the epidermis contain the following:
melanocytes, which produce melanin, a pigment that gives skin its color and protects it from the damaging effects of ultraviolet radiation
keratinocytes, which produce keratin, a water-repellent protein that gives the epidermis its tough, protective quality
dermis
composed of a thick layer of skin that contains collagen and elastic fibers, nerve fibers, blood vessels, sweat and sebaceous glands, and hair follicles
subcutaneous tissue
composed of a fatty layer of skin that contains blood vessels, nerves, lymph, and loose connective tissue filled with fat cells
Functions of the integument
protection
intact skin prevents invasion of the body by bacteria
thermoregulation
intact skin facilitates heat loss and cools the body when necessary through the following processes:
production of perspiration which assists in cooling the body through evaporation
production of vasodilatation to increase the blood supply to the skin surface which assists in facilitating heat loss from the body through radiation and conduction
intact skin prevents heat loss and warms the body when necessary through the following processes:
prevention of the production of perspiration which inhibits cooling the body through evaporation
production of vasoconstriction to decrease the blood supply to the skin surface which assists in preventing heat loss from the body through radiation and conduction
production of gooseflesh by contraction of the arrector pili muscles attached to hair follicles in order to stand skin hairs on end thus allowing them to entrap a thick layer of "insulator air" next to the skin which assists in preventing heat loss from the body through radiation and conduction
fluid and electrolyte balance
intact skin prevents the escape of water and electrolytes from the body
vitamin D synthesis
intact skin facilitates the synthesis of vitamin D through conversion of the initial precursor of the vitamin to the second precursor of the vitamin in the skin by the sun's ultraviolet rays
sensation
intact skin assists the body in receiving sensory stimuli from the environment by activation of skin receptors for pain, touch, pressure, and temperature
psychosocial
intact skin contributes to a positive body image
Classification of wounds
by cause
intentional
involves a wound that is the result of planned therapy
unintentional
involves a wound that is the result of unexpected trauma
by status of skin integrity
open
involves a break in skin integrity or mucous membrane
closed
involves no break in skin integrity or mucous membrane
by severity of injury
superficial
involves only the epidermal layer of skin
penetrating
involves penetration of the epidermal and dermal layers of skin and deeper tissues or organs
by degree of contamination
clean
uninfected wounds in which no inflammation is encountered and the respiratory, gastrointestinal, genital, and/or urinary tracts are not entered
clean/contaminated
uninfected wounds in which no inflammation is encountered but the respiratory, gastrointestinal, genital, and/or urinary tract have been entered
contaminated
open, traumatic wounds or surgical wounds involving a major break in sterile technique that show evidence of inflammation
infected
old, traumatic wounds containing dead tissue and wounds with evidence of a clinical infection (e.g., purulent drainage)
by depth
partial-thickness
involves only the epidermal and dermal layers of skin
full-thickness
involves the epidermal and dermal layers of skin, subcutaneous tissue and, possibly, muscle and bone
by descriptive qualities
laceration
involves tearing apart of tissues resulting in irregular wound edges
abrasion
involves scraping or rubbing the surface of the skin by friction
contusion
involves a blow from a blunt object resulting in swelling, discoloration, bruising, and/or eccymosis
incision
involves cutting the skin with a sharp instrument
puncture
involves penetration of the skin and, often, the underlying tissues by a sharp instrument
Phases of wound healing
defensive (inflammatory) phase
begins immediately after injury, lasting 3-4 days, and consists of two major processes: hemostasis and inflammation
hemostasis
vasoconstriction of severed blood vessels
aggregation of platelets along damaged blood vessel walls to form a platelet plug
invasion of fibrin fibers into the platelet plug to form a fibrin clot
contraction of the fibrin clot to express fluid (serum) out of the clot and form a scab which provides external protection from invasion by microorganisms
inflammation
process of inflammation
injury to tissues resulting in secretion of multpile products of inflammation into the blood stream from the injured tissues, e.g.: histamine, bradykinin, serotonin, prostaglandins
leads to a clinical sign of inflammation:
dolor (pain)
vasodilation of local blood vessels, resulting in excess local blood flow and an increase in nutrients to the injured tissues
leads to two clinical signs of inflammation:
calor (warmth)
rubor (hyperemia)
increased permeability of capillaries, resulting in leakage of large quantities of fluid into the interstitital spaces to cushion the injured tissues and dilution of the concentration of microorganisms or toxic products that may have entered the injured tissues
leads to a clincial sign of inflammation:
tumor (swelling or edema)
chemotaxis (attraction of white blood cells [WBCs] to injured tissues), resulting in migration of tissue macrophages, neutrophils, and monocytes to the injured tissues and phagocytosis of microorganisms and debris in the injured tissues
1st line of defense in wound healing by WBCs
description
tissue macrophage invasion of the injured tissue
white blood cell(s) involved
tissue macrophage
time of response
minutes after injury
response
fixed tissue macrophages break away from their attachments near injured tissues, migrate to the injured tissues, and begin phagocytosis of about:
100 bacteria per macrophage
large diameter debris
2nd line of defense in wound healing by WBCs
description
neutrophil invasion of the injured tissues
white blood cell(s) involved
neutrophil
time of response
few hours after injury
response
within the first hours after inflammation begins, circulating neutrophils migrate to the injured tissues and begin phagocytosis of about:
5 - 20 bacteria per neutrophil
small diameter debris
3rd line of defense in wound healing by WBCs
description
second macrophage invasion of the injured tissues
white blood cell(s) involved
monocyte
time of response
several days after injury
response
stored monocytes in the bone marrow are mobilized into the blood stream, migrate to the injured tissues where they attach themselves to the injured tissues, spend around 8 hours or more maturing into a tissue macrophages, and after the maturation process, begin phagocytosis of about:
100 bacteria per macrophage
large diameter debris
4th line of defense in wound healing by WBCs
description
increased proliferation of granulocytes and monocytes by the bone marrow
white blood cell(s) involved
granulocyte
monocyte
time of response
3 - 4 days after injury
response
bone marrow greatly increases production of granulocytes and monocytes which are mobilized into the blood stream to injured tissues and begin phagocytosis
encouragement of actions to avoid further injury to or inflammation of the injured tissues
leads to a clinical sign of inflammation:
functiolaesa (loss of function)
walling off injured tissues and lymphatics from remaining tissues to delay spread of microorganisms or toxic products
proliferative phase
begins 3 or 4 days after injury, lasting up to 3 weeks
macrophages stimulate the migration of fibroblasts to the wound to synthesize collagen and ground substance (proteogylcan)
the collagen and proteogylcan synthesized by fibroblasts forms a scaffold, or framework, for final repair of the wound and can be felt as a "healing ridge" under the suture
the more collagen and ground substance added to the scaffold, or framework, the greater the tensile strength of the wound
macrophages also stimulate the formation of "buds" in capillaries surrounding the wound that grow into new blood vessels (angiogensis) and reestablish blood flow across the wound
the collagen and proteogylcan deposits and capillary "buds" form the new granulation tissue in the wound, which is translucent red, fragile, and bleeds easily
proliferation of epithelial cells across the wound as granulation tissue matures
maturation phase
begins after 21 days after injury, lasting months and even years
firbroblasts continue to synthesize collagen and proteoglycan and add it to the wound to increase its tensile strength
the collagen and proteogylcan fibers, laid haphazardly during the proliferative phase, reorganize into a more orderly structure
Types of wound drainage
serous exudate
consists chiefly of serum derived from blood and serous membranes of the body
sanguineous
consists of serum and red blood cells
purulent
consists of serum and pus (leukocytes, liquefied living and dead bacteria, dead tissue debris)
Types of wound healing
healing by primary intention
characterized by:
approximated (closed) skin edges
minimal granulation tissue
early suturing
minimal scarring
minimal tissue loss
rapid healing
minimal risk of infection
healing by secondary intention
characterized by:
open skin edges
extensive granulation tissue
no suturing
extensive scarring
extensive tissue loss
delayed healing
extensive risk of infection
healing by tertiary intention
characterized by:
open skin edges that are sutured later
moderate granulation tissue
delayed suturing
moderate scarring
moderate tissue loss
delayed healing
moderate risk of infection
Complications of wound healing
hemorrhage
persistent arterial and/or venous bleeding from a wound
usually occurs in the first 24 - 48 hours after a wound
signs/symptoms:
decreased blood pressure
rapid thready pulse
restlessness
diaphoresis
clammy, pale, cold skin
oliguria
infection
invasion of a wound by microorganisms
usually occurs by the 2nd to 11th day after a wound
signs/symptoms:
increased temperature
tenderness and pain around wound site
increased WBCs
inflamed wound edges
purulent drainage
dehiscence
partial or total rupturing of a wound
usually occurs by the 4th to 5th day after a wound
in abdominal wounds, it often occurs after a sudden strain, such as coughing, vomiting, sitting up in bed, and sneezing which makes it important to support the abdomen during these activities
signs/symptoms:
partially or totally disrupted (open) wound edges
appreciable increase in discharge of serosanguineous drainage from the wound
sensation that "something gave or let go"
increased sanguineous drainage
evisceration
protrusion of the internal viscera and/or organs through a ruptured wound
usually occurs by the 3rd to 5th day after a wound
in abdominal wounds, occurs after a sudden strain, same as a dehiscence
signs/symptoms:
totally disrupted (open) wound edges
appreciable increase in discharge of serosanguineous drainage from the wound
sensation that "something gave or let go"
protrusion of viscera and/or organs through a ruptured wound
medical emergency with surgical repair necessary; if occurs, place sterile towels soaked in sterile normal saline solution over extruding tissue to decrease chance of invasion by microorganisms and drying
fistula
abnormal passage between two organs or between an organ and the outside of the body
name of the fistula designates the site of the abnormal communication; e.g., a rectovaginal fistula is an abnormal opening between the rectum and vagina that permits feces to enter the vagina
signs/symptoms:
chronic drainage (type depends on communication)
skin breakdown (if fistula opens to the skin, and the drainage is gastric or intestinal in nature, so contains digestive enzymes)
Factors affecting wound healing
impaired circulation and oxygenation, e.g.:
advanced age
anemia
peripheral vascular disorders
diabetes mellitus
smoking
obesity
chronic lung disease
malnourishment
results in inadequate intake of nutrients essential for wound healing, e.g.:
protein: essential for building new tissue
vitamin A: essential for collagen synthesis and epithelialization
B complex vitamins: act as cofactors in enzyme reactions necessary for wound healing
vitamin C: essential for collagen synthesis, capillary formation, resistance to infection
vitamin K: essential for synthesis of prothrombin which assists in hemostasis
zinc, copper, and iron: essential for collagen synthesis
drugs and therapies that effect the inflammatory response, blood clotting, and cell mitosis, e.g.:
adrenocorticoidsteriods
acetylsalicylic acid (aspirin)
warfarin sodium (Coumadin)
heparin
antineoplastics (anti-cancer drugs)
radiation
contamination and infection
Decubitis ulcers
any skin lesion caused by unrelieved pressure that results in damage to underlying tissues
etiology of decubitus ulcers
localized ishcemia
tissue is caught between two hard surfaces
blood cannot reach the tissues
cells are deprived of oxygen and nutrients
waste products of metabolism accumulate in cells
cells eventually die
friction
rubbing the patients skin against the sheets when pulling him/her up in bed
shearing
adherence of the patient's skin to the sheets, while layers of subcutaneous tissues slide in the direction of the body movement and underlying capillaries are severed, when a patient slides down in bed
risk factors for development of decubitus ulcers
immobility
results in the inability to relieve pressure by changing position
malnutrition
results in weight loss which reduces the padding between the skin and bones
hypoproteinemia
precipitates the formation of edema which makes skin more susceptible to pressure
fecal and urinary incontinence
causes maceration (tissue softened by prolonged wetting and soaking) which makes skin more susceptible to pressure
decreased mental status
results in inability to respond to pain from increased pressure by changing position
diminished sensation
results in inability to respond to pain from increased pressure by changing position
advanced age
results in the following:
loss of lean body mass
thinning of the epidermis
decreased elasticity of the skin due to loss of collagen,
decreased vascularity
reduced skin turgor
increased dryness and scaliness
decreased pain perception
common locations of decubitus ulcers
over bony prominences while lying in a supine position in bed, e.g.:
back of head (occipital bone), scapulae, elbows (olecranon process), sacrum, heels (calcaneous)
over bony prominences while lying in a lateral position in bed, e.g.:
side of head (parietal and temporal bones), ear, shoulder (acromion process), ilium, greater trochanter, knee (medial and lateral epicondyle)
over bony prominences while lying in a prone position in bed, e.g.:
over bony prominences while sitting in a wheelchair, e.g.:
shoulder blade, sacrum and coccyx, ischial tuberosity, posterior knee, foot
staging/grading decubitus ulcers
stage/grade one
nonblancable erythema of intact skin
stage/grade two
partial-thickness skin loss involving the epidermis and/or dermis
presents clinically as an abrasion, blister, or shallow crater
stage/grade three
full-thickness skin loss involving damage or necrosis of subcutaneous tissue that may extend down to, but not through, underlying fascia
presents clinically as a deep crater with or without undermining of adjacent tissue
stage/grade four
full-thickness skin loss with extensive destruction, tissue necrosis or damage to the muscle, bone, or supporting structures, such as a tendon or joint capsule
The nursing process and wounds
assessing
treated wounds
pain
appearance of wound, e.g.:
approximation of wound edges
color of the wound and surrounding area
signs of dehiscence or evisceration
wound drainage
sutures and staples, e.g.:
types of sutures in treated wounds
absorbable
used to attach tissues beneath the skin
made of material that disappears in several days
nonabsorbable
used to attach tissues of the skin
made of a variety of nonabsorbable materials, e.g.:
silk, cotton, linen, wire, nylon, Dacron
methods of suturing in treated wounds
plain, interrupted sutures
consists of one pass per stitch
pierces the near edge of the incision line (e.g., right) and exits the opposite edge of the incision line (e.g., left)
each stitch is tied and knotted separately
mattress, interrupted sutures
consists of two passes per stitch
lst pass
pierces the near edge of the incision line (e.g., right) and exits the opposite edge of the incision line (e.g., left)
2nd pass
pierces the new near edge of the incision line (e.g., left) and exits the new opposite edge of the incision line (e.g., right)
each stitch is tied and knotted separately
plain, continuous sutures
one thread runs in a series of stitches in the plain suturing method (one pass per stitch) and is tied only at the beginning and end of the run
mattress, continuous sutures
one thread runs in a series of stitches in the matress suturing method (two passes per stitch) and is tied only at the beginning and end of the run
retention sutures
very large sutures used in addition to skin sutures that attach underlying tissues of fat and muscle as well as skin and are used to support in incisions in obese individuals when healing may be prolonged
drains and tubes
penrose
provides a sinus tract
t-tube
provides for bile drainage
Jackson-Pratt
provides for decreased dead space by collecting drainage
hemovac
provides for decreased dead space by collecting drainage
decubitus ulcers
location of the decubitus ulcer
estimation of the stage/grade of the decubitus ulcer
color of the decubitus ulcer wound bed
presence or absence and location of any necrosis or eschar in the decubitus ulcer wound bed
condition of the decubitus ulcer wound margins
integrity of the skin surrounding the decubitus ulcer
clinical signs of infection
dimensions of the decubitus ulcer
length and width
place a baggie, saran wrap, or a disposable measuring guide over the decubitus ulcer
trace over the decubitus ulcer using a black marker
depth
insert a sterile cotton-tipped applicator at a 90 degree angle into the deepest part of the decubitus ulcer
mark the point on the sterile cotton-tipped applicator that is even with the surrounding skin surface of the decubitus ulcer
remove the sterile cotton-tipped applicator
measure the depth against a centimeter ruler
tunneling
insert a saline-moistened sterile cotton-tipped applicator under the edges of the decubitus ulcer
apply gentle pressure with the sterile cotton-tipped applicator
assess for any abnormal pathways
remove the sterile cotton-tipped applicator
measure the depth of penetration against a centimeter ruler
laboratory data
albumin levels of less than 3.5 mg/dL
total lymphoctye count of less than 1,000/mm3
coagulation studies
wound cultures
Diagnosing
impaired skin integrity
Planning
goal/expected outcomes:
the patient will exhibit unimpaired skin integriy, as evidenced by:
approximated wound edges
a decrease in (or absence of) wound drainage
a decrease in (or absence of) inflammation of skin surrounding the wound
intact skin surrounding the wound
absence of wound odor
a decrease in length, width, and depth of the wound
presence of granulation tissue in the wound
Implementing
support wound healing
provide sufficient nutrition and fluids
prevent wound infections
proper positioning
prevent decubitus ulcers
provide sufficient nutrtion and fluids
turn every 2 hours
use positioning devices to keep weight off bony prominences, e.g.:
gel flotation pads
special polyvinyl, silicone, or silastic pads filled with a gelatinous substance similar to fat
sheepskins
special devices make of natural or artificial sheepskin
pillows and wedges
pillows or foam wedges used to raise a body part off the bed surface
heel protectors
special devices, such as sheepskin boots, padded splints, or foam wedges, that limit pressure on the heels
prevent shearing forces by limiting the amount of time the head of the bed is elevated (if possible)
prevent friction sources by using a trapeze or bed linens to transfer and/or change a patient's position
use pressure-relieving support surfaces, e.g.:
egg crate mattress
special mattress made of polyurethane foam resembling an eggcrate
foam mattress
special mattress made of polyurethane foam that molds to a patient's body
alternating pressure mattress
special mattress composed of a number of cells in which the pressure is alternately increased and decreased
water bed
special mattress filled with temperature controlled water
air-fluidized (AF) bed (static high air loss [HAL] bed)
special bed consisting of a mattress in which forced, temperature controlled air is circulated around millions of tiny silicone-coated beads producing a fluid-like movement that also helps prevent maceration of the patient's skin in the following ways:
body moisture penetrates the sheet
the beads soak up the moisture
the air forces the moisture laden beads away from the patient
the beads fall to the bottom of the mattress
the sheet is rapidly dried
static low air loss (LAL) bed
special bed consisting of many air-filled cushions divided into four or five sections in which separate control panels permit each section to be inflated to a different level of firmness in such a way that pressure can be reduced on bony prominences but increased under other body areas for support
active or second generation low air loss (LAL) bed
special bed similar to the static low air loss (LAL) bed except that it also gently pulsates or rotates side to side in order to stimulate capillary blood flow and facilitate movement of pulmonary secretions
cleanse the skin routinely and whenever soiling occurs
keep linens clean, dry, and wrinkle-free
maintain skin hygeine
dress wounds
purposes of dressings
provide physical, pyschological, and aesthetic comfort
remove necrotic, or dead, tissue
prevent, eliminate, or control infection
absorb drainage
maintain a moist wound environment
protect the wound from further injury
protect the skin surrounding the wound
types of dressings
type of dressing used depends on:
the location, size, and type of the wound
the amount of exudate
whether the wound requires debridement, is infected, or has sinus tracts
frequency of dressing change, ease or difficulty of dressing application, and cost
type of dressings available:
guaze
2 x 2
4 x 4
Sof-WickÒ drain sponges
Surgi-padÒ
abdominal (ABD) pad
transparent films
clear, polyurethane film with an adhesive backing
examples
Op-siteÒ, TegadermÒ, BiocclusiveÒ, ACU-dermÒ
purpose/properties
provide moist wound environment
no absorptive properties
occlusive
waterproof
semi-permeable to oxygen
provide good wound visualization
uses
partial-thickness wounds
Stage I-II pressure ulcers
protective cover for areas exposed to friction
to secure a gauze pad or another type of absorbent dressing in place
hydrocolloid wafers
hydroactive wafers (usually tan) with an adhesive backing
examples
DuoDermÒ, ComfeelÒ, TegabsorbÒ, RestoreÒ
purpose/properties
provide moist wound environment
absorb moderate amount of wound exudate
occlusive
waterproof
impermeable to oxygen
autolytic debridement
interact with wound exudate to form a hydrated gel that is thick, yellow and malodorous when removed
uses
partial-thickness wounds
Stage I-II pressure ulcers
hydrogels
hydroactive polymer sheets, granules, or gels that are nonadhesive and require a secondary dressing to secure