1500 milliliters of chyme per day pass through the large intestine
all but 100 milliliters of water is reaborbed from the 1500 milliliters of chyme that pass through the large intestine per day
secrete mucus into the large intestine
protect the large intestine from bacterial activity
act as an adherent for the feces
eliminate the products of digestion
flatus
feces
three types of large intestine movments
haustral churning
back and forth movement in the haustra that helps mix the chyme
peristalsis
wave like movement that propels the chyme forward
mass peristalsis
wave of powerful muscular contraction over large areas of the large intestine
Process of fecal elimination
expulsion of feces from the rectum and anus
feces enters the rectum
stimulates a signal that initiates peristaltic waves in the descending and sigmoid colon and the rectum
peristaltic waves force feces into the anus
as the peristaltic waves approach the anus, the internal spinchter is inhibited from closing (opened involuntarily)
individual senses need to defecate and finds a toilet
the individual relaxes the external anal spinchter (opened voluntarily)
defecation occurs
Factors influencing fecal elimination
age and development
children
desire to control daytime fecal elimination occurs when a child becomes aware of the following (usually around two 1/2 years old):
discomfort of soiled diaper
sensation that indicates need for elimination of feces
nurses can become involved in a childs bowel training in the following ways:
continuing the bowel training program established at home while the child is in the hospital
educating parents on methods for successful bowel training, such as providing their child with the following:
clothes that can be removed independently
a personal toilet seat
sufficient time to eliminate feces
a consistent, relaxed atmosphere
praise for successful behavior while avoiding punishment for unsuccessful behavior
a non-stressful period in which to initiate toilet-training
elderly
changes in the elderly that can effect fecal elimination include the following:
decreased esophageal and gastrointestinal (GI) peristalsis
can result in slower passage of chyme in the large intestine, more water being reabsorbed from the chyme, and accumulation of air and gas and constipation
poor dentition and decreased digestive enzyme (e.g., pytalin, pepsin, trypsin, lipase) and gastric acid secretion
can result in partially digested food which may result in indigestion
decreased tone of the abdominal, pelvic, and thigh muscles
can result in a decreased amount of intra-abdominal pressure that can be exerted during defecation
decreased anal spinchter tone
can result in urgency to defecate or fecal incontinence
diet
insufficient fiber (undigested residue) in the diet can effect fecal elimination
does not provide the fecal volume necessary to stretch the walls of the large intestine, stimulate peristalsis, and initiate the defecation reflex which may result in accumulation of air and gas and constipation
foods high in fiber
raw fruits (e.g., apples, oranges)
cooked fruits (e.g., prunes, apricots)
greens (e.g., spinach, kale, cabbage)
raw vegetables (e.g., celery, zucchini)
whole grains (e.g., cereal, bread)
certain foods can effect fecal elimination by encouraging the following:
gas (e.g., cabbage, onions, cauliflower, bananas, and apples)
diarrhea (e.g., bran, prunes, figs, chocolate, and alcohol)
constipation (e.g., cheese, pasta, eggs, and lean meat)
certain food conditions can effect fecal elimination, such as the following:
lactose intolerance
increases peristalsis and mucus secretion in the large intestine which results in rapid passage of chyme through the large intestine, less water being reabsorbed from the chyme, and diarrhea
food allergies
reduces digestion of food elements which increases peristalsis in the large intestine, results in rapid pasage of chyme through the large intestine, less water being reabsorbed from the chyme, and diarrhea
fluid
inadequate fluid intake (less than 2,000 to 3,000 milliliters per day) or excessive fluid output can effect fecal elimination
can result in slower passage of chyme in the large intestine, more water being reabsorbed from the chyme, and constipation
activity
inadequate activity can effect fecal elimination
inhibits peristalsis which results in slower passage of chyme in the large intestine, more water being reabsorbed from the chyme, and accumulation of air or gas and constipation
decreases the tone of the abdominal, pelvic and thigh muscles which can result in a decreased amount of intra-abdominal pressure that can be exerted during defecation
psychologic factors
certain emotional states can effect fecal elimination
anxiety/stress activates the sympathetic portion of the autonomic nervous system (ANS) which inhibits large intestine peristalsis, increases the tone of the internal and external anal spinchters, and may potentially lead to accumulation of air and gas and constipation
certain diseases of the large intestine are thought to have a psychological component
e.g., ulcerative colitis, Chrons disease
life-style
certain life-style behaviors can effect fecal elimination by delaying defecation which allows water to be reabsorbed from the chyme in the large intestine and constipation, such as the following:
not defecating at a regular time
e.g., after breakfast to take advantage of the gastrocolic and duodenocolic reflexes
ignoring the urge to defecate
unavailability of toilet facilities
embarrassment about odors and noises
lack of privacy
not exerting intra-abdominal pressure while defecating
e.g., assuming a squatting position, contracting the abdominal, pelvic, and thigh muscles, and leaning forward
medications
certain medications can effect fecal elimination, such as the following:
those that decrease peristalsis which results in slower passage of chyme in the large intestine, more water being reabsorbed from the chyme, and accumulation of air and gas and constipation, e.g.:
narcotic analgesics (e.g., morphine sulphate)
antidepressants (e.g., isocarboxazid [Marplan])
anticholinergics (e.g., benzotropine [Cogentin]
those that increase peristalsis in the large intestine which results in faster passage of chyme in the large intestine, less water being reabsorbed from the chyme, and diarrhea, e.g.:
antibiotics (e.g., penicillin)
it is thought that the normal flora of bacteria in the large intestine keep the pathogenic flora under control
antibiotics (especially broad-spectrum) can suppress the normal flora of bacteria in the large intestine allowing pathogenic bacteria to multiply and produce toxins irritating to large intestine which may result in diarrhea (suprainfection)
ferrous sulfate
irritates the mucosa of the large intestine which may result in diarrhea
laxatives
normal defecation reflexes are so inhibited that habitual users require larger or stronger doses to be able to defecate
diagnostic procedures
certain diagnostic procedures can effect fecal elimination in the following ways:
barium remaining in the colon after a barium enema can harden producing constipation or possibly an impaction
cleansing enemas given to empty the colon of fecal material prior to certain diagnostic procedures can interrupt normal fecal elimination
anesthesia and surgery
general anesthetic agents can effect fecal elimination in the following way:
use of general anesthetic agents may cause temporary slowing or cessation of peristalsis which may result in accumulation of air and gas and constipation
surgery on the large intestine can effect fecal elimination in the following way:
manipulation of the large intestine during surgery may cause temporary cessation of peristalsis for 24 - 48 hours (paralytic ileus) which can result in accumulation of air and gas and constipation
surgical alterations of gastrointestinal anatomy can effect fecal elimination in the following ways:
gastrectomy
the loss of the reservoir function of the stomach results in dumping of food into the duodenum too rapidly for proper absorption and diarrhea
colon resection
the reduced size of the large intestine results in less area for reabsorption of water from the chyme in the large intestine and diarrhea
pathologic conditions
certain pathologic conditions can effect fecal elimination
particularly those that do the following:
decrease or eliminate sensory stimulation necessary for competence of the external anal spinchter and defecation
certain pathologic conditions of the colon (ulcerative colitis, irritable bowel syndrome, Chron's disease) can effect fecal elimination in the following ways:
the inflammation and/or ulceration in the large intestine increases peristalsis which results in the faster passage of chyme in the large intestine, decreased reabsorption of water from the chyme, and diarrhea
pain
certain conditions which cause pain upon defecation can effect fecal elimination, such as the following:
hemorrhoids
anal fissures
Common fecal elimination problems
constipation
the state in which an individuals pattern of elimination is characterized by dry, hard stool that results from a delay in passage of food residue
related factors (etiology)
less than adequate fluid/dietary intake
less than adequate physical activity
lack of privacy
emotional disturbances
stress
change in daily routine
chronic use of medications and enemas
metabolic problems
neuromuscular impairment
musculoskeletal impairment
weak abdominal musculature
pain on defecation
diagnostic procedures
medication side-effects
pregnancy
defining characteristics
decreased frequency
painful defecation
abdominal distention
abdominal pain
rectal pressure
appetite impairment
headache
dry, hard stool
straining at stool
palpable mass
interventions
cleansing enema
cathartics
bulk-forming
uses synthetic or natural polysaccharides and cellulose derivatives to absorb water into the fecal mass to add bulk
plantago seed (psyllium seed)
lubricant
prevents excessive absorption of water from the feces thus delaying drying of the feces
mineral oil
Haley's MO
wetting agent
lowers the surface tension of the feces which helps water and fatty substances to penetrate the feces
doucasate sodium (Colace)
stimulant
irritates the intestinal mucosa causing increased peristalsis and rapid expulsion of feces
castor oil
cascara sagrada
bisacodyl (Dulcolax, Bisco-lax)
senna
saline
uses salts, which are poorly absorbed in the intestine, to draw fluid into the intestine which increases fecal bulk and possibly lubricates the feces
magnesium sulfate (Epsom salts)
magnesium hydroxide (Milk of Magnesia)
magnesium citrate
danger of constipation is that it causes straining at defecation which can result in the following:
put pressure on abdominal or perineal sutures which may precipitate wound dehisence or eviseration
promote the development of hemorroids
elicit the valsalva manuever (holding breath while straining) which can be dangerous in clients with heart, respiratory, or brain injuries because it does the following:
slows the heart rate by stimulating the vagus (10th cranial) nerve and, consequently:
decreases the return of blood to the heart
may precipitate dysrhythmias
increases intra-abdominal pressure
which concomittantly increases venous, intrathoracic, and intracranial pressure
fecal impaction (etiology)
mass or collection of hardened feces, wedged in the folds of the rectum and possibly higher, accompanied by inability to pass a normal stool and seepage of liquid fecal material from the anus
related factors
same as for constipation
defining characteristics
inability to pass a stool
seepage of liquid fecal material from the anus
palpable mass
interventions
digital exam (if impaction is suspected)
may be too far up to feel
may need a physicians order since it can precipitate the valsalva manuever
oil retention enema
cleansing enema 2 - 4 hours later
digital removal of impaction (if the above are not ineffective)
danger of fecal impacation
same as for constipation
if allowed to persist, may require surgery for removal of a fecalith
diarrhea
the state in which an individual experiences a change in normal bowel habits characterized by the frequent passage of loose, fluid, unformed stools
related factors (etiology)
gastrointestinal disorders
metabolic/endocrine disorders
nutritional disorders
change in dietary intake
infectious processes
tube feedings
adverse effects of medications
high stress level
defining characteristics
abdominal pain
urgency
cramping
increased frequency
increased frequency of bowel sounds
loose, liquid stools
interventions
decrease or eliminate food intake to decrease peristalsis
antidiarrheals
adsorbents
coat the walls of the large intestine, absorbing the bacteria or toxins causing diarrhea, and passing them out with the feces
bismuth subsalicylate (Pepto-Bismol)
activated charcoal (Charcocaps)
attapulgite (Kaopectate)
anticholinergics
not conclusively known to decrease peristalsis, but used to prevent spasm and cramping associated with acute diarrhea (in a sufficient dose)
atropine
hyoscyamine
hyoscine
homatropine methylbromide
opiates
reduce large intestinal tone and peristalsis, reduce pain, and relieve tenesmus (rectal spasms)
tincture of opium
paregoric
codeine
intestinal flora modifiers
Lactobacillus organisms that reestablish the normal flora of bacteria in the large intestine which is though to help suppress the growth of diarrhea-producing pathogenic bacteria in the large intestine
lactobacillus acidophilus and lactobacillus bulgaricus (Lactinex)
lactobacillus acidophilus in sodium carboxymethylcellulose (Bacid)
danger of diarrhea
can precipitate serious fluid and electrolyte imbalances
especially in infants and small children
fecal incontinence
the state in which an individual experiences a change in normal bowel habits characterized by involuntary passage of stool
related factors
gastrointestinal disorders
neuromuscular disorders
colostomy
loss of rectal spinchter control
impaired cognition
defining characteristics
involuntary passage of stool
interventions
bowel training
determine the clients usual bowel habits and factors that facilitate or hinder normal defecation
design a plan to promote normal elimination habits, e.g.,
a fluid intake of 2,000 - 3,000 mililiters per day
increased fiber in the diet
increased exercise
privacy
empty the large intestine
use a purgative if necessary
begin daily bowel training program
administer a daily mixture of cathartics
have client drink a hot drink before the daily scheduled defecation time
insert a cathartic suppository or apply digital stimulation after hot drink
assist client to the toilet or on to a bedpan
have client (or nurse) apply intra-abdominal pressure during attempt to defecate
set time limit for defecation
fecal incontinence pouch
a pouch that is secured around the anal opening and may or may not be attached to drainage
flatulence (tympanities)
presence of excessive flatus (air or gas) in the large intestine in the intestinal tract
related factors (etiology)
accumulation of air or gas in the large intestine through swallowing air, the action of bacteria on the chyme in the large intestine, and/or diffusion of gas from the bloodstream into the large intestine which cannot escape from the mouth (belched) or the anus (as flatus)
defining characteristics
abdominal distention
abdominal pain
interventions
carminative enema
return flow or Harris flush enema
rectal tube
Characteristics of feces
normal feces
color: adult = brown; infant = yellow
consistency: formed, soft, semisolid, moist
shape: cylindrical (contour of rectum), about one inch in diameter (adults)amount: varies with diet, but about 100 - 400 grams per day
odor: aromatic; affected by ingested food and clients own bacterial flora
constituents: small amounts of undigested roughage, sloughed dead bacteria and epithelial cells, fat, protein; dried constituents of digestive juices (e.g., bile), inorganic matter (e.g., calcium, phosphates)
abnormal feces
color: clay or white; black or tarry; red or pale; orange or green
consistency: hard, dry, diarrhea
shape: narrow; pencil-shaped; or string-like feces
amount: more than 400 grams per day
odor: pungent
constituents: pus, mucus, parasites, blood, large amounts of fat, foreign objects
Diagnostic studies
indirect visualization of the GI tract
flat-plate of the abdomen
description
plain radiograph of the abdomen to determine gross anatomic features of the organs of the abdomen
patient preparation
none
follow-up
none
barium swallow
description
ingestion of a contrast medium that allows indirect visualization of the pharynx and esophagus
patient preparation
NPO after midnight the evening before the test
follow-up
drink fluids to eliminate the barium
mild laxative or stool softener if necessary
educate patient that stools will be chalky white or lighter in color for several days
upper GI series and small bowel follow through (SBFT)
description
ingestion of a contrast medium that allows indirect visualization of the lower esophagus, stomach, duodenum, ileum, and jejunum
patient preparation
NPO after midnight the evening before the test
educate the patient that he/she will be on a rotating x-ray table
educate the patient that he/she will need to assume many positions
follow-up
drink fluids to eliminate the barium
mild laxative or stool softener if necessary
educate patient that stools will be chalky white or lighter in color for several days
barium enema
description
administration of a contrast medium enema to allow indirect visualization of the large intestine
patient preparation
low residue diet 2 days prior to the test
clear liquid diet the evening before the test
NPO after midnight the evening before the test
potent laxative the night before the test (e.g., magnesium citrate)
oral liquid preparation the night before the test (e.g., GoLytely)
cleansing enema the night before the test
follow-up
drink fluids to eliminate the barium
mild laxative or stool softener if necessary
cleansing enema if necessary
educate patient that stools will be chalky white or lighter in color for several days
liver scan (renography)
description
injection of a radionuclide medium into a vein and monitoring its uptake in the liever to provide gross information about liver blood flow, structure, and function
patient preparation
ascertain that a consent form is signed
follow-up
monitor vital signs according to agency protocol
wear rubber gloves when discarding the patient's urine for 24 hours
wash the gloved hands with soap and water after discarding the patient's urine for 24 hours
wash the ungloved hands with soap and water after discarding the patient's urine for 24 hours
educate the patient to wash hands with soap and water after each voiding for 24 hours
gastric analysis
description
measurement of hydrochloric acid and pepsin secretion by the stomiach through insertion of a nasogastric tube and collection of samples of gastric secretions at 15 minute intervals for 1 hour
patient preparation
NPO at least 12 hours prior to the test
educate the patient to not chew gum or smoke cigarettes for 6 hours prior to the test
educate the patient that a nasogastric tube will be inserted into his/her stomach
educate the patient that samples of gastric secretions will be collected, measured, and labled every 15 minutes for an hour
educate the patient that the nasogastric tube will be removed after the procedure
follow-up
none
direct visualization of the GI tract
proctoscopy
description
viewing of rectum with a proctoscope
patient preparation
liquid diet 24 hours prior to the test
cleansing enema or Fleets enema prior to test
laxative the eveing before the test
follow-up
check for s/s of perforation (pain, bleeding)
sigmoidoscopy
viewing of the sigmoid colon with a sigmoidoscope
patient preparation
proctoscopy
follow-up care
same as for proctoscopy
colonoscopy
definition
viewing of the large intestine with an endoscope
patient preparation
liquid diet 24 hours prior to the test
NPO after midnight the night of the test
potent laxative the night before the test (e.g., GoLytely)
follow-up care
check vital signs per policy
siderails up
check for s/s of perforation (pain, bleeding)
esophophagogastroduodenoscopy
definition
viewing of the esophogus, stomach, and upper duodenum with an endoscope
patient preparation
NPO after midnight the night of the test
follow-up care
check vital signs per policy
siderails up
NPO for 1 - 2 hours or until gag reflex returns
check for s/s of perforation (pain, bleeding)
blood tests
prothrombin time
collection of a specimin of blood to determine prothrombin time
normal findings:
11 - 12.5 seconds
significance of abnormal findings:
increased values, possible:
deficiency in prothrombin, fibrinogen, or extrinsic factors
liver disease
vitamin K deficiency
calcium
collection of a specimin of blood to determine serum calcium levels
normal findings:
9 - 10.5 mg/dL or
2.25 - 2.75 mmol/L
significance of abnormal findings:
decreased values, possible
malabsorption
renal failure
acute pancreatitis
potassium
collection of a specimin of blood to determine serum potassium levels
normal findings:
3.5 mEq/L
3.5 mmol/L
significance of abnormal findings:
decreased values, possible
vomiting
gastric suctioning
diarrhea
drainage from intestinal fistulas
serum electrophoresis
collection of a specimin of blood to determine serum electrophoresis
normal findings:
total protein = 6 - 8 g/dL
albumin = 3.5 - 5 g/dL
alpha1-globulin = 0.1 - 0.4 g/dL
aplha2-globulin = 0.5 - 1g/dL
beta globulin = 0.7 - 1.2 g/dL
gamma globulin = 0.5 - 1.6 g/dL
significance of abnormal findings:
decreased values, possible
hepatic disease
GI disorders
peptic ulcer
acute cholecystitis
malabsorption
AST, SGOT
collection of a specimin of blood to determine serum AST, SGOT levels
normal findings:
8 - 20 units/L or
5 - 40 IU/L
significance of abnormal findings:
increased values, possible
viral hepatitis
cirrhosis
acute pancreatitis
malabsorption
other liver damage
ALT, or SGPT
collection of a specimin of blood to determine serum ALT or SGPT levels
normal findings:
5 - 35 IU/L
significance of abnormal findings:
increased values, possible
hepatitis
cirrhosis
LDH
collection of a specimin of blood to determine serum LDH levels
normal findings:
45 - 90 units/L or
115 - 225 IU/L or
0.4 - 1.7 mmol/L
significance of abnormal findings:
damaged liver caused by hepatitis or hepatocellular disorders
alkaline phosphatase
collection of a specimin of blood to determine serum alkaline phosphatase levels
normal findings:
30 - 90 IU/L
significance of abnormal findings:
hepatic disease
biliary obstruction
bilirubin (total serum)
collection of a specimin of blood to determine serum bilirubin levels
normal findings:
0.1 - 1 mg/dL or
5.1 - 17 mmol/L
significance of abnormal findings:
hemolysis
biliary obstruction
hepatic damage
bilirubin (conjugated, direct)
collection of a specimin of blood to determine serum bilirubin levels
normal findings:
0.1 - 0.3 mg/dL or
1.7 - 5. mmol/L
significance of abnormal findings:
biliary obstruction
bilirubin (unconjugated, indirect)
collection of a specimin of blood to determine serum bilirubin levels
normal findings:
0.2 - 0.8 mg/dL or
3.4 - 12 mmol/L
significance of abnormal findings:
hemolysis
hepatic damage
ammonia
collection of a specimin of blood to determine serum ammonia levels
normal findings:
15 - 110 mg/dL or
47 - 65 mmol/L
significance of abnormal findings:
hepatic disease (e.g., cirrhosis)
d-Xylose absorption
collection of a specimin of blood to determine serum d-Xylose levels
normal findings:
25 - 40 mg/dL in 2 hours or
3.5 g in 5 hours or
5 g in 24 hours
significance of abnormal findings:
malabsorption in the small intestine
serum amylase
collection of a specimin of blood to determine serum amylase levels
normal findings:
80 - 150 Somogyi units/dL or
56 - 90 IU/L or
25 - 125 units/L
significance of abnormal findings:
acute pancreatitis
serum lipase
collection of a specimin of blood to determine serum lipase levels
normal findings:
0 - 110 units/L or
0 - 417 units/L
significance of abnormal findings:
acute pancreatitis
cholesterol
collection of a specimin of blood to determine serum cholesterol levels
normal findings:
150 - 200 mg/dL or
3.9 - 6.5 mmolL
significance of abnormal findings:
increased values, possible,
pancreatitis
biliary obstruction
decreased values, possible
liver cell damage
urine tests
bilirubin
collection of a specimin of urine to determine urinary bilirubin levels
normal findings:
negative
significance of abnormal findings:
biliary obstruction
cirrohsis
hepatitis
urobilinogen
collection of a specimin of urine to determine urinary urobilinogen levels
normal findings:
0.1 - 1 unit/mL
significance of abnormal findings:
hepatitis
cirrhosis
amylase
collection of a specimin of urine to determine urinary amylase levels
normal findings:
various levels, depending on unit of measure
significance of abnormal findings:
acute pancreatitis
pancreatic obstruction
stool specimins
collection of a specimin of stool to ascertain the presence of normal/abnormal constituents of stool
stool for occult blood
collection of a specimin of stool to ascertain the presence/absence of occult (hidden) blood in the stool
normal findings:
negative
presence, diagnostic for
carcinoma
peptic ulcer
ulcerative colitis
stool for ova and parasites
collection of a specimin of stool to ascertain the presence/absence of ova or parasites in the stool
normal findings:
negative
presence, diagnostic for
infection
stool for fecal fat
collection of a specimin of stool to ascertain the amount of fat in the stool
normal findings:
2 - 5 g/24 hours with normal diet
increased values, possible
Chron's disease
malabsorption syndrome
pancreatic disease
Bowel diversion ostomies
jejunostomy
diversion of chyme from the small intestine by surgically implanting the jejunum onto the skin surface and creating an opening (stoma)
ileostomy
diversion of chyme from the small intestine by surgically implanting the ileum onto the skin surface and creating an opening (stoma)
cecostomy
diversion of feces from the large intestine by surgically implanting the cecum onto the skin surface and creating an opening (stoma)
ascending colostomy
diversion of feces from the large intestine by surgically implanting the ascending colon onto the skin surface and creating an opening (stoma)
transverse loop colostomy
diversion of feces from the large intestine by surgically implanting the transverse colon onto the skin surface and creating an opening (stoma)
transverse double-barreled colostomy
diversion of feces and mucous from the large intestion by surgically implanting the proximal transverse colon onto the skin surface and creating an opening (stoma) (the colostomy) and surgically implanting the distal transverse colon onto the skin surface and creating an opening (stoma) (the mucous fistula)
descending colostomy
diversion of feces from the large intestine by surgically implanting the descending colon onto the skin surface and creating an opening (stoma)
sigmoid colostomy
diversion of feces from the large intestine by surgically implanting the sigmoid colon onto the skin surface and creating an opening (stoma)